Nolvadex and hair loss. why?

I'm using 20 mg a day and I did not know that, now I got a problem since I'm losing my hair these days.

Maybe we had a misunderstanding. Nolvadex cap is 60mg. I think you are taking nolvadex, that is 20mg/cap.

Indeed, my bad, thanks. I'm taking tamoxifene while on Abombs'>Anadrol.

Re: Nolvadex and hair loss. why?

Posted by: duchaine

A lot of guys repost hair loss while they take Nolvadex.
Why does it happen?
a) because reduced estogen;
b)because increased DHT;
c) because Nolvadex produces some metabolites that can be bad for hair.

I hope the right answer is B, so, taking propecia, I can fix the problem.
Every opinion and suggestion is well accepted!
thank you

Anybody else having hair problems with Nolvadex?..I was thinking of trying Nolvadex with my winter training but starting to believe it's not worth it..My hair is already thin and the feedback I'm reading seems inconclusive to its T benefits..Am I wrong?

same problem. even lasts several weeks after you stop.

Probably because estrogens tend to protect the scalp hair follicle.

Exp Dermatol. 2002 Aug;11(4):376-80.Click here to read Links
17alpha-estradiol induces aromatase activity in intact human anagen hair follicles ex vivo.
Hoffmann R, Niiyama S, Huth A, Kissling S, Happle R.

Department of Dermatology, Philipp University, Deutschhausstrasse 9, D-35033 Marburg, Germany.

For topical treatment of androgenetic alopecia (AGA) in women, solutions containing either estradiol benzoate, estradiol valerate, 17beta- or 17alpha-estradiol are commercially available in Europe and some studies show an increased anagen and decreased telogen rate after treatment as compared with placebo. At present it is not precisely known how estrogens mediate their beneficial effect on AGA-affected hair follicles. We have shown recently that 17alpha-estradiol is able to diminish the amount of dihydroTestosterone(DHT) formed by human hair follicles after incubation with testosterone, while increasing the concentration of weaker steroids such as estrogens. Because aromatase is involved in the conversion of testosterone to estrogens and because there is some clinical evidence that aromatase activity may be involved in the pathogenesis of AGA, we addressed the question whether aromatase is expressed in human hair follicles and whether 17alpha-estradiol is able to modify the aromatase activity. Herewith we were able to demonstrate that intact, microdissected hair follicles from female donors express considerably more aromatase activity than hair follicles from male donors. Using immunohistochemistry, we detected the aromatase mainly in the epithelial parts of the hair follicle and not in the dermal papilla. Furthermore, we show that in comparison to the controls, we noticed in 17alpha-estradiol-incubated (1 nM) female hair follicles a concentration- and time-dependent increase of aromatase activity (at 24 h: 1 nM = +18%, 100 nM = +25%, 1 micro M = +57%; 24 h: 1 nM = +18%, 48 h: 1 nM = +25%). In conclusion, our ex vivo experiments suggest that under the influence of 17alpha-estradiol an increased conversion of testosterone to 17beta-estradiol and androstendione to estrone takes place, which might explain the beneficial effects of estrogen treatment of AGA.

nope no problems here